Balance and Vestibular Disorders Explained

Dizziness is hard to describe and harder to ignore. Sometimes it feels like spinning. Sometimes it feels like balance is "just a bit off." For many people it comes and goes with posture - getting out of bed, for example. A smaller number of people have a more persistent sense of being slightly unsteady, even sitting down. Finding words that capture the experience is genuinely difficult, and that difficulty reflects the fact that the balance system draws on several different inputs at once. What goes wrong varies depending on which part is involved.

Balance and Vestibular Disorders Explained

It helps to separate three common sensations that are often grouped together. Vertigo is the illusion of movement, usually spinning. Presyncope is the feeling that you might faint, often linked to blood pressure or cardiovascular causes. Disequilibrium is a sense of unsteadiness or imbalance, particularly when walking. People can experience more than one of these at the same time. Identifying which is dominant often points toward the underlying cause.

Most balance disorders fall into a small number of recognisable patterns based on when the dizziness happens, how long it lasts, what seems to trigger it, and whether anything else accompanies it. That history is usually what points toward a diagnosis well before any testing is done.

The conditions below cover the most common causes seen in audiology and ear, nose and throat practice. Some are straightforward to resolve. Some are managed over time rather than cured. The connection between hearing and balance is not always obvious - both functions share the same inner ear structure, which is why an audiologist is often one of the first clinicians involved and why a hearing assessment frequently forms part of the diagnostic picture. If something in the list below sounds familiar, it is worth following up rather than waiting

Common balance and vestibular conditions

Benign paroxysmal positional vertigo (BPPV)

BPPV is the most common cause of vertigo. It produces brief, sudden episodes of spinning - usually lasting less than a minute - triggered by specific head movements such as rolling over in bed, looking up, or bending forward. Between episodes, most people feel entirely normal, which is one of the reasons it can feel bewildering. The cause is mechanical: small calcium carbonate crystals (otoconia) that normally sit in one part of the inner ear balance organs become dislodged and migrate into one of the structures involved in sensing head movements - semicircular canals. Once there, they create an abnormal signal during movement that the brain interprets as spinning.

Diagnosis involves a specific manoeuvre called the Dix-Hallpike, which places the head in a position that provokes the characteristic eye movement (nystagmus) if BPPV is present. The pattern of that eye movement - its direction, how quickly it appears, and how quickly it settles - tells the clinician which of the three semicircular canals is involved. The posterior canal is most commonly the source, though the horizontal and anterior canals can also be affected. Treatment is a repositioning sequence called the Epley manoeuvre, which guides the displaced crystals back to where they belong. Resolution rates are high, often after one or two treatments. It is common to feel off-balance for a few days afterward, but this usually settles on its own. BPPV can recur, particularly in older adults, but responds well to the same approach each time (Bhattacharyya et al., 2017).

Vestibular neuritis

Vestibular neuritis arrives suddenly and hard. Most people describe waking with severe vertigo, nausea, and a pronounced sense of being pulled or thrown, often to one side. Moving makes it worse. The episode typically peaks within hours and then gradually settles over days to weeks, though a residual unsteadiness can persist for longer. Hearing is preserved, which distinguishes it from labyrinthitis, where the adjacent cochlea is also affected.

The cause is thought to be viral inflammation of the vestibular nerve. During the acute phase, the brain receives mismatched signals from the two ears, which drives the vertigo and instability. Over time the brain recalibrates to work with this asymmetry, a process called central compensation. Vestibular rehabilitation is the most meaningful intervention for recovery. Corticosteroids may improve recovery on vestibular testing, but this does not consistently translate to better day-to-day function (Strupp et al., 2004; Leong et al., 2021).

Meniere's disease

Meniere's disease produces episodic attacks of vertigo lasting from twenty minutes to several hours, accompanied by fluctuating hearing loss, tinnitus that sounds like ocean-waves crashing, and a sensation of pressure or fullness in the affected ear. The combination is characteristic. The unpredictability of attacks is often what patients find hardest to manage. The underlying mechanism is thought to involve excess inner ear fluid pressure (endolymphatic hydrops), though the exact relationship to symptoms is still being studied.

Diagnosis relies on the pattern of symptoms over time and audiometric evidence of fluctuating low-frequency hearing loss. In early stages, hearing often recovers between episodes. Over time, the loss can become more permanent. Audiological monitoring is an important part of care. Management ranges from dietary and medical approaches through to intratympanic therapy in more persistent cases (Lopez-Escamez et al., 2015).

Vestibular schwannoma (acoustic neuroma)

A vestibular schwannoma is a benign, slow-growing tumour arising from the vestibular nerve. Despite the name, it most often presents with hearing-related symptoms:

gradual one-sided hearing loss, reduced speech clarity, and sometimes persistent tinnitus. Vertigo is less common because the slow growth allows the brain to compensate.

On hearing tests, asymmetry in thresholds combined with disproportionately reduced speech understanding can raise suspicion. MRI with contrast is the definitive investigation. Early identification matters, as management options depend on tumour size and growth rate at detection.

Vestibular migraine

Vestibular migraine is one of the most common causes of episodic dizziness and also one of the most frequently missed. It produces recurrent episodes of vertigo or dizziness lasting minutes to hours, often with light sensitivity, sound sensitivity, or headache - though headache may be absent. That absence can make the diagnosis less obvious.

There is no single diagnostic test. Diagnosis is based on the pattern of episodes over time using established clinical criteria. Between episodes, vestibular testing is often normal or shows only minor non-specific findings. Hearing is usually not permanently affected. Management follows migraine principles, including trigger management and preventative treatment where needed (Lempert et al., 2012).

Persistent postural-perceptual dizziness (PPPD)

PPPD is a chronic functional vestibular condition. The inner ear may be functioning normally, but the way the brain processes balance information has shifted. It is characterised by persistent non-spinning dizziness or unsteadiness lasting at least three months, typically worse when upright, with movement, or in visually busy environments.

PPPD often follows an acute vestibular event such as BPPV or neuritis. The brain adopts a more visually dependent and vigilant processing style and does not fully return to baseline. Vestibular testing is often normal or shows only minor residual findings. Treatment focuses on vestibular rehabilitation, gradual re-exposure to provoking environments, and addressing associated anxiety where present (Staab et al., 2017).

Bilateral vestibulopathy

Bilateral vestibulopathy involves reduced or absent function of both vestibular systems. It is less common but clinically distinctive. Symptoms include oscillopsia, where the visual world appears to bounce with head movement, and marked imbalance that worsens in low light or on uneven ground.

Causes include certain antibiotics (particularly gentamicin), bilateral Meniere's disease, autoimmune conditions, and in some cases a gradual decline with no clearly identified cause. Diagnosis involves testing how well the eyes and inner ear work together during head movement - a response called the vestibulo-ocular reflex (VOR) - along with other balance measures. Rehabilitation focuses on teaching the system to make the most of what remains, since once both sides are affected, recovery of lost function is limited (Strupp et al., 2017).

When to seek urgent care

Most causes of dizziness are benign, but some symptoms need urgent medical assessment. These include new double vision, difficulty speaking, weakness or numbness in the face or limbs, a new severe headache, sudden hearing loss with vertigo, or continuous severe vertigo that does not begin to settle. These features may indicate a central or vascular cause and should not be ignored.

The importance of history

Across all of these conditions, the most useful diagnostic tool is a careful account of what is actually happening. The timing of episodes, how long they last, what provokes them, and what accompanies them narrow the picture substantially before any test result is available.

If you have been referred for a balance or vestibular assessment, or if something in this description sounds familiar, the booking link is below. An assessment at hear. includes basic functional vestibular evaluation where indicated, along with the full audiometric picture and a written referral where onward investigation or specialist input is needed. Most vestibular conditions are benign, many are highly treatable, and even the persistent ones can be managed effectively with the right approach.


Bhattacharyya, N., Gubbels, S. P., Schwartz, S. R., Edlow, J. A., El-Kashlan, H., Fife, T., Holmberg, J. M., Mahoney, K., Hollingsworth, D. B., Roberts, R., Seidman, M. D., Steiner, R. W., Do, B. T., Voelker, C. C., Waguespack, R. W., Corrigan, M. D., & Robertson, P. J. (2017). Clinical practice guideline: Benign paroxysmal positional vertigo (update). Otolaryngology-Head and Neck Surgery, 156(3 Suppl), S1-S47. https://doi.org/10.1177/0194599816689667

Lempert, T., Olesen, J., Furman, J., Waterston, J., Seemungal, B., Carey, J., Bisdorff, A., Versino, M., Evers, S., & Newman-Toker, D. (2012). Vestibular migraine: Diagnostic criteria. Journal of Vestibular Research, 22(4), 167-172. https://doi.org/10.3233/VES-2012-0453

Leong, K. J., Lau, T., Stewart, V., & Canetti, E. F. (2021). Effectiveness of corticosteroids in treating adults with acute vestibular neuritis: A systematic review and meta-analysis. Otolaryngology-Head and Neck Surgery, 165(2), 255-266. https://doi.org/10.1177/0194599820976840

Lopez-Escamez, J. A., Carey, J., Chung, W. H., Goebel, J. A., Magnusson, M., Mandala, M., Newman-Toker, D. E., Strupp, M., Suzuki, M., Trabalzini, F., & Bisdorff, A. (2015). Diagnostic criteria for Meniere's disease. Journal of Vestibular Research, 25(1), 1-7. https://doi.org/10.3233/VES-150549

Staab, J. P., Eckhardt-Henn, A., Horii, A., Jacob, R., Strupp, M., Brandt, T., & Bronstein, A. (2017). Diagnostic criteria for persistent postural-perceptual dizziness (PPPD). Journal of Vestibular Research, 27(4), 191-208. https://doi.org/10.3233/VES-170622

Strupp, M., Brandt, T., & Muller, A. (2004). Corticosteroids for acute vestibular neuritis. New England Journal of Medicine, 351(4), 354-361. https://doi.org/10.1056/NEJMoa033280

Strupp, M., Kim, J. S., Murofushi, T., Straumann, D., Jen, J. C., Rosengren, S. M., Della Santina, C. C., & Kingma, H. (2017). Bilateral vestibulopathy: Diagnostic criteria consensus document of the Classification Committee of the Barany Society. Journal of Vestibular Research, 27(4), 177-189. https://doi.org/10.3233/VES-170619